2013 — Histamine; Histamine receptors; Histamine H4 receptor, human; Atopic Histamin förmedlar sina cellfysiologiska verkningar via fyra G- Till professionella histaminproducerande celler räknas MC, basofiler, ECL celler och.
The ECL-cell histamine mobilization was independent of the concentrations of Ca2+ in the perfusion medium (0-3.4 mmol/l Ca2+). In one experiment, histamine mobilization in response to gastrin
3). The ECL-cell histamine mobilization was independent of the concentrations of Ca2+ in the perfusion medium (0-3.4 mmol/l Ca2+). In one experiment, histamine mobilization in response to gastrin Histamine from the ECL cells being mainly located at the base of the glands (7), may reach the parietal cell not only by the paracrine route but also via the capillaries ( 18). Thus, the ECL cell via histamine release plays a central role in the regulation of gastric acid secretion (Figure). The ECL cell ▪ Abstract Enterochromaffin-like (ECL) cells are neuroendocrine cells in the gastric mucosa that control acid secretion by releasing histamine as a paracrine stimulant. Enterochromaffin-like (ECL) cells are neuroendocrine cells in the gastric mucosa that control acid secretion by releasing histamine as a paracrine stimulant.
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som är inblandad i att bygga cellväggar och förbättrar gallblåsans, I magsäckens slemhinna finns s.k. ECL-celler som frisätter histamin I centrala nervsystemet metyleras histaminet av Histamine N-methyltransferase
av L Kölby · 1999 — ECL cell histamine gastrin. CCK-B/gastrin receptor. Mastomys natalensis midgut carcinoid 5-HT somatostatin receptor vesicular monoamine transporter Neuroendocrine cells in the glands of the GASTRIC MUCOSA. They produce HISTAMINE and peptides such as CHROMOGRANINS.
Breast implants: Update on TGA monitoring of anaplastic large cell lymphoma (27 September for allergic reactions due to higher than expected amounts of histamine. Waiver and Consent Form FICSD:ECL,FICSD:LAW LABEL:ACADEMIC
Gastrin itself is secreted by cells in the epithelium of the stomach, but travels to ECL cells via the blood. Together, histamine and gastrin are primary positive regulators of acid secretion from the parietal cell. Enterochromaffin-like (ECL) cells are neuroendocrine cells in the gastric mucosa that control acid secretion by releasing histamine as a paracrine stimulant. The antral hormone gastrin and the neural messenger pituitary adenylyl cyclase–activating peptide (PACAP) potently stimulate histamine synthesis, storage, and secretion by ECL cells.
Thus, the ECL cell via histamine release plays a central role in the regulation of gastric acid secretion (Figure). The ECL cell may also produce other mediators.
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The ECL cells in the oxyntic mucosa produce, store and secrete histamine and pancreastatin (a chromogranin A‐derived peptide) in response to gastrin (Håkanson et al., 1986; Prinz et al., 1993; Chen et al., 1994, 1996; Lindström et al., 1997) and there is much evidence to suggest that the acid‐stimulating effect of gastrin is mediated by ECL‐cell histamine (Waldum et al., 1991 Current students New students International Desk Academic matters & support IT services & support Careers Service 2008-07-01 dependent on histamine or Reg-1 protein [19] or another mediator from the ECL cell. The ECL cell has prolongations approaching the parietal cell in a synaptic-like manner [20]. It should also be added that the ECL cell produces calbindin [21], and at least upon gastrin stimulation, the alfa-glycoprotein hormone [22].
Histamine builds up and strays from its typical behavior. Our journals promote pharmacology in all its forms by disseminating the latest high quality research in our peer reviewed scientific journals.
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Vol. 16, No. 2, 1988 ECL CELL HYPERPLASIA AND NEOPLASIA 275 TABLE 1.-Characteristics of carcinoid tumors in the fundic mucosa of rats in the carcinogenicity study. Lesion Compound size Locally (mglkg) Rat no. Sex Fate (mm) invasive BL-6341 8 M ST 1.0 No (300)b 19 M ST 0.7 No 36 M DW 2.OC Yes
They secrete histamine in response to circulating gastrin. Gastric submucosal microdialysis has been used to study ECL-cell histamine mobilization in awake rats. Histamine in the stomach occurs in endocrine cells (so-called enterochromaffin-like (ECL) cells), mast cells, and neurons.
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Håkanson, ECL Cell Histamine Mobilization Studied byGastric Submucosal. Microdialysis in Awake Rats:Methodological Considerations., Artikel i tidskrift,.
The antral hormone gastrin and the neural messenger pituitary adenylyl cyclase–activating peptide (PACAP) potently stimulate histamine synthesis, storage, and secretion by ECL cells. Histamine is stored in secretory vesicles via V-type ECL-cell histamine plays an important role in the regulation of gastric acid secretion, particularly in response to gastrin stimulation. The time-course responses of ECL cells to gastrin include Gastric ECL Cells Studies on isolated ECL cells showed that gastrin potently induces histamine secretion from these cells within minutes of incubation. This gastrin effect is hence classified as an acute effect on ECL cell function. In enriched, uncultured ECL cells gastrin-induced stimulation was two- to threefold. After 48 h of short- Abstract: The ECL cells are endocrine/paracrine cells in the acid-producing part of the stomach. They secrete histamine in response to circulating gastrin.
1997-12-01
This is in line with the previously demonstrated weak stimulatory effect of NMU‐25 and secretin on isolated ECL cells (Lindström et al., 1997). a-FMHdirectly impaired parietal cell activity [46]. Basalacid secretion wasreducedbyapproximately 60percentin aX-FMH-treatedrats compared to vehicle-treated rats, suggesting an involvement of ECL-cell histamine in basal acid secretion. Howeverthe H2 antagonist ranitidine inhibited basal acid secretion by about 85 percent. 2001-12-01 1997-12-01 The histamine response to electrical vagal stimulation reflects the effect of circulating gastrin rather than a direct action of the vagus on the ECL cells. Vagal denervation was accompanied by an impaired histamine response to food intake, probably reflecting the right‐ward shift of the serum gastrin concentration–histamine response curve. The ECL cells in the oxyntic mucosa produce, store and secrete histamine and pancreastatin (a chromogranin A‐derived peptide) in response to gastrin (Håkanson et al., 1986; Prinz et al., 1993; Chen et al., 1994, 1996; Lindström et al., 1997) and there is much evidence to suggest that the acid‐stimulating effect of gastrin is mediated by ECL‐cell histamine (Waldum et al., 1991 Current students New students International Desk Academic matters & support IT services & support Careers Service 2008-07-01 dependent on histamine or Reg-1 protein [19] or another mediator from the ECL cell.
T1 - The vagus regulates histamine mobilization from rat stomach ECL cells by controlling their sensitivity to gastrin. AU - Norlén, Per. AU - Ericsson, Peter. AU - Kitano, M. AU - Ekelund, Mats. AU - Håkanson, Rolf. PY - 2005. Y1 - 2005 NMU‐25 almost qualified as a stimulator of ECL cell histamine mobilization (90% increase, integrated response) while secretin exerted weaker stimulatory effects (35% increase).